What causes an increase in uric acid?

Causes of a high uric acid level in the blood include: Diuretics (water retention relievers) Drinking too much alcohol. Drinking too much soda or eating too much of foods that contain fructose, a type of sugar.

mayoclinic.org - High uric acid level Causes - Mayo Clinic

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A high uric acid level can be the result of the body making too much uric acid, not getting rid of enough of it or both.

Causes of a high uric acid level in the blood include:

Diuretics (water retention relievers) Drinking too much alcohol Drinking too much soda or eating too much of foods that contain fructose, a type of sugar Genetics also known as inherited traits High blood pressure (hypertension) Immune-suppressing drugs Kidney problems Leukemia Metabolic syndrome Niacin, also called vitamin B-3 Obesity Polycythemia vera Psoriasis A purine-rich diet, high in foods such as liver, game meat, anchovies and sardines Tumor lysis syndrome — a rapid release of cells into the blood caused by certain cancers or by chemotherapy for those cancers People having chemotherapy or radiation treatment for cancer might be monitored for high uric acid levels. Causes shown here are commonly associated with this symptom. Work with your doctor or other health care professional for an accurate diagnosis. There is a problem with information submitted for this request. Review/update the information highlighted below and resubmit the form. From Mayo Clinic to your inbox Sign up for free, and stay up to date on research advancements, health tips and current health topics, like COVID-19, plus expertise on managing health. Email ErrorEmail field is required ErrorInclude a valid email address Learn more about Mayo Clinic’s use of data. To provide you with the most relevant and helpful information, and understand which information is beneficial, we may combine your email and website usage information with other information we have about you. If you are a Mayo Clinic patient, this could include protected health information. If we combine this information with your protected health information, we will treat all of that information as protected health information and will only use or disclose that information as set forth in our notice of privacy practices. You may opt-out of email communications at any time by clicking on the unsubscribe link in the e-mail. Subscribe! Thank you for subscribing! You'll soon start receiving the latest Mayo Clinic health information you requested in your inbox. Sorry something went wrong with your subscription Please, try again in a couple of minutes Retry

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Gout progresses through four clinical phases: asymptomatic hyperuricemia, acute gouty arthritis, intercritical gout (intervals between acute attacks) and chronic tophaceous gout.

aafp.org - Gout and Hyperuricemia - AAFP

The use of low-dose colchicine as prophylaxis for the prevention of gouty arthritis was first described in 1936.27 It is common practice among rheumatologists to administer prophylactic or low-dose colchicine (from 0.6 mg to 1.2 mg) at the same time urate-lowering drug therapy is initiated, but this regimen has not been widely embraced by primary care physicians.28,29 Prophylactic therapy is quite effective in patients who tolerate colchicine and is 85 percent effective in preventing acute attacks.30 Colchicine should be used for prophylaxis only with concurrent use of urate-lowering agents. Colchicine alone does not alter urate deposition or tissue damage.31 Low-dose colchicine is used for prophylaxis until the serum urate concentration is stable at the desired level and the patient has been free from acute gouty attacks for three to six months. There is a risk for an acute gouty flare-up when prophylaxis is discontinued. However, most patients are able to remain on urate-lowering agents alone. If patients do not tolerate daily doses of colchicine, a low daily dose of a selected NSAID can be used instead.

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